Cells | Free Full-Text | Identification of Transcription Factors Responsible for a Transforming Growth Factor-β-Driven Hypertrophy-like Phenotype in Human Osteoarthritic Chondrocytes
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Local inhibition of TGF-β1 signaling improves Th17/Treg balance but not joint pathology during experimental arthritis | Scientific Reports
Biology | Free Full-Text | Transcription Factors in Cartilage Homeostasis and Osteoarthritis
Alpha 5 Integrin Mediates Osteoarthritic Changes in Mouse Knee Joints | PLOS ONE
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TGF-β is a potent inducer of Nerve Growth Factor in articular cartilage via the ALK5-Smad2/3 pathway. Potential role in
IJMS | Free Full-Text | Osteoarthritis-Related Inflammation Blocks TGF-β's Protective Effect on Chondrocyte Hypertrophy via (de)Phosphorylation of the SMAD2/3 Linker Region
PDF) Differential Role of Transforming Growth Factor-beta in an Osteoarthritic or a Healthy Joint
The high affinity ALK1-ligand BMP9 induces a hypertrophy-like state in chondrocytes that is antagonized by TGFβ1
Selective Inactivation of a Human Neuronal Silencing Phosphatase by a Small Molecule Inhibitor | ACS Chemical Biology
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